ALTHOUGH a blood chemical analysis reveals air excessive sodium carbonate content which the related free phosphorus deficiency, a chemical condition necessary for the production of any pathology in a localized tissue site as a result of deposition of the blood carbonates,. it must be fully realized that the nature of the tissue-pathology is determined by the cellular conditions existing at the carbonate deposition site. The basic factors determining carbonate deposition from the blood and tissue fluids are the presence of the excessive blood carbonates and the mechanical stasis in the circulating fluids requisite to permit deposition from the circulating stream.
Venous and lymph stasis determine the localized body- sites in which deposition can occur. Injury, traumatic or otherwise, determines lymph stasis and therefore predisposes to deposition. If, however, the lymph and venous circulation contain efficiency of free phosphorus, and of necessity in such a case an absence of carbonates, circulatory stasis cannot result in those pathologies dependent on the chemical action of the deposited carbonates or the caustic hydrates evolved by the hydrolysis of the carbonates. Excessive blood carbonates are essential for localized cancer cellular growth, being the source from which the caustic carcinogenic agent arises.
An excessive blood carbonate content must always be a danger signal-local carbonate deposition may not have already resulted, but a continuance of the processes which have caused the excessive blood carbonate state must inevitably end in conditions through which any degree of lymph or venous stasis will determine carbonate deposition. According to the tissue-site of deposition the- nature of the resulting pathological lesion is evolved. What determine the nature of the so-called benign fibroid tumour, the renal, thyroid, ovarian, pancreatic and genital-cord cysts, and finally the cancer or malignant growth, is not the blood chemical carbonate condition but the localized tissue-site into which the carbonates are deposited. The establishment of excessive blood carbonates by blood analysis, or more simply by an analysis of the urinary excretion through which the body endeavours to rid itself of its carbonate incubus, does not differentiate between local benign or malignant pathologies. The blood and urine carbonate-analysis establishes, however, whether malignancy can be present in any localized pathology when it is correlated with the anatomical knowledge of the site in which the suspected malignant growth is present. Cancer cell evolution and growth being due to the continuous local action of caustic hydrates, formed after the deposition from the circulating fluids, of carbonates, can only occur at body-sites in which carbonate hydrolysis can occur. Hydrolysis of carbonates is impossible in a serious lined cavity which gives no exit for the gaseous carbon dioxide. If, however, a continuous vent should occur in such a serous sac, forming the deposition reservoir for excessive blood carbonates, hydrolysis of the carbonates can and does occur with the formation of the carcinogenic hydrate and the evolution of its cancer cell pathology. If carbonate deposition should occur, as it does in the muscle wall of the female uterus, the carbonates retain their chemical state and the resulting increasing or progressive fibrosis is entirely the reaction to the carbonate deposition. This fibrosis once begun with an excessive blood carbonate state becomes progressive because the primary fibrosis determines continuous lymph and venous stasis with its sequela -continuous carbonate deposition.