I wouldnt feel as though I had presented a case before the society if I hadnt given all the scientific laboratory data. We have to consider that. We know that to be a symptom-monger professionally doesnt mean very much-at least it doesnt to me. I must bring proof and in that way I educate myself and I understand and perceive what the homoeopathic remedy does.

I wish to submit a report of two cases of meningitis that came under my service at the Childrens Department of the Womens Homoeopathic Hospital of Philadelphia. I will give the history and complete laboratory reports copied from the charts and records at the hospital. I feel it is necessary to present the original laboratory reports and chart history to help to confirm the diagnosis of a serious case when a cure is made with a high potency of a homoeopathic remedy.

There are various clinical forms of meningitis, as you possibly know. These are classified according to the different organisms found in the spinal fluid and blood. The first case discussed here has a confirmed diagnosis of influenzal meningitis. This child was admitted to the Childrens building through the Dispensary, on January 9, 1935. A colored girl, age 6 years; her chart history is as follows:.

FAMILY HISTORY:Mother living and well. Two other children living and well. No history of cardio-renal disease, epilepsy or diabetes. Father died recently of tuberculosis.

PAST PERSONAL HISTORY: No childhood diseases, no operations, no history of injuries; patient well until present illness.

PRESENT ILLNESS: One week ago, patient began to complain of cough, coryza, and became very feverish. There was marked prostration; several times she vomited, which was projectile in character. She developed severe convulsions and was taken to the Hospital. The provisional diagnosis made by the interne was bronchopneumonia.

PHYSICAL EXAMINATION:Black female child in convulsions,m semiconscious, apparently very toxic. Pupils react sluggishly to light and accommodation; marked convergent strabismus in left eye. Chest; increased area of bronchovesicular breathing; increased tactile fremitus; dullness, particularly over right mammary region. Subcrepitant rales scattered over both sides of chest. Respirations very labored and rapid.

Marked dyspnoea. Frequent short, tight, apparently painful cough, Heart: Fair myocardial tone, no audible murmurs. Neurological examination: Marked retraction of head with rigidity of neck, giving a positive Brudzinskis sign. Pupils dilated; convergent squint. Positive Kernig sign more marked on left side. Positive Babinski, knee jerks increased, general hyperaesthesia.

I did a cisternal tap and collected 40 mill. of cloudy fluid under marked pressure.

LABORATORY REPORT:Spinal Fluid-Cloudy fluid, turbid. 30 c.c. examined. Globulin present, 2 plus. Sugar negative. Cell count 180. M.E. Pus, 4 plus. Culture of the spinal fluid showed a distinct strain of influenzal bacilli.

(Signed), GEORGE HOPP, M.D., Pathologist X-RAY OF CHEST. Report: Examination of the chest shows bronchopneumonia involving both lungs. No evidence of pleural effusion or encysted fluid.

LABORATORY REPORT: Blood: The complete blood analysis will not be given here. All counts were done by the Shilling method. The hemogram revealed: January 10, 1935: Degenerative shift to the left. Prognosis grave. January 11, 1935: Degenerative shift to the left. Prognosis fatal.

These are the reprints of the history of the case and the laboratory diagnostics.

The symptoms for the selection of the remedy were entirely objective as this young child was unable to furnish us with any reliable subjective symptoms. On admission, the temperature was 104 plus respiration 50, pulse 160. On the signs and symptoms related, the interne prescribed Belladonna which was allowed to act for three days, with only a slight amelioration of temperature.

When the patient developed opisthotonos, marked squint, became practically unconscious, with all the former neurological findings increased, I said another cisternal drainage, and took some of the fluid and potentized it according to the Fincke method, and began giving it January 12. Within 48 hours, we began to notice a most spectacular change in the patient. The temperature began to drop, respirations became easier, the child regained consciousness, and the pulse improved in quality.

Within four days, her temperature was normal. At the end of a week, the spinal fluid showed very little pus, the cell count was greatly reduced, and by January 21, the neurological findings cleared up. The strabismus had cleared up, were the cervical opisthotonos had disappeared, and there were no Babinski or Kernig sings.

The case came to a standstill for about a week, when Sulphur was given. Another spinal tap gave a perfectly clear fluid, normal cell count, with no pus or organisms present. There now remained no symptoms except anorexia, extreme emaciation, which had been rapid and pronounced, a few rales in the chest which had not cleared up, and an occasional cough. Arsenic iod. was prescribed here.

William B. Griggs