“Those who hoped that the administration of insulin to a. diabetic might lead to the cure of the underlying disease. are apparently to be disappointed,. As a result of. observations lasting from 11 to 18 months on give selected. cases of diabetes mellitus, no evidence has been obtained by Dr.G.A.Harrison of even a partial remission of the disease. The daily intake of carbohydrate, protein, fat, and calories remained fixed throughout. The dose of insulin was adjusted to the fixed diet according to the results of estimation of the blood-sugar content. All five patients needed as much or slightly more insulin at the end of the period of observation”.

We quote this merely to emphasize the fact, that however valuable insulin may be, it is nevertheless not a cure, but is to be looked upon in the light of substitutive medicine. The diabetic patient will still need to be put upon a suitable diet and so far as we homoeopaths are concerned, will need to be prescribed for as an individual.

There are no remedies for diabetes, but there are many remedies for patients who have diabetes; quite another matter. Cure remains, as it always has, an elusive thing and difficult to achieve.

Iodin and Goitre-In the London Lancet for “February 6th, the leading article by Sir James Berry, B.S., London: F.R.C.S., Eng., is entitled, “Some Clinical Aspects of Simple Goitre, with Remarks on its Causation.” In speaking of the varieties of goitre Sir James says:

“If we leave aside malignant disease, acute inflammation,. and certain of the rare forms of thyroid enlargement, we are left with two main varieties of goitre. The first is characterized by excess of the cellular elements of the gland and by diminution or absence of colloid. This is exophthalmic goitre in its various forms. It appears to be essentially a hypertrophy of the gland, although this is probably not the whole explanation of the condition.

“The second variety is that of simple endemic goitre, and the first point that I want to emphasize is, that simple endemic goitre is not a hypertrophy of the gland. It is essentially a degeneration. The enlargement of the gland in the earliest stages in which I have been able to observe it, consists primarily epithelial elements. In this respect it differs entirely from the goitre of Graves disease, in which, as mentioned above, the enlargement is due to an increase in the cellular elements of the gland. This misuse of the term hypertrophy as applied to simple goitre, is very widely spread in literature at the present day”.

Further on, in combating the prevalent idea that a lack of iodin is the cause of goitre, the author states:

The most convincing single proof of the baselessness of the lack of Iodine theory is afforded by what occurred in connexion with goitre at Sanawar in North India, investigated by Colonel McCarrison and fully reported by him in the British Medical journal of June 7th, 1924. At a large school in an institution at the place, where a high incidence of goitre had been present for many years,he found in 1913 that over 80 percent, of the children were affected. By his advice a new and pure water-supply was introduced in 1918. In 1922 goitre was reported to have disappeared from the school. In the autumn of 1923, when he revisited the school, the incidence of goitre was only 2.2 per cent., or no greater than the incidence of thyroid enlargement among school children residing in a non-goitrous district such as Delhi.

He was assured by the principal that no changes had been made in the food. Samples of the new water-supply were analysed by the chemical examiner to the Punjab Government, who reported that it contained no iodine. Not satisfied with this, Colonel McCarrison caused 25 gallons of water to be evaporated to dryness after the addition to it of sodium carbonate. The residue thus obtained was sent to the chemical examiner for estimation of its iodine content. He reported that it contained no iodine.

Allan D. Sutherland
Dr. Sutherland graduated from the Hahnemann Medical College in Philadelphia and was editor of the Homeopathic Recorder and the Journal of the American Institute of Homeopathy.
Allan D. Sutherland was born in Northfield, Vermont in 1897, delivered by the local homeopathic physician. The son of a Canadian Episcopalian minister, his father had arrived there to lead the local parish five years earlier and met his mother, who was the daughter of the president of the University of Norwich. Four years after Allan’s birth, ministerial work lead the family first to North Carolina and then to Connecticut a few years afterward.
Starting in 1920, Sutherland began his premedical studies and a year later, he began his medical education at Hahnemann Medical School in Philadelphia.
Sutherland graduated in 1925 and went on to intern at both Children’s Homeopathic Hospital and St. Luke’s Homeopathic Hospital. He then was appointed the chief resident at Children’s. With the conclusion of his residency and 2 years of clinical experience under his belt, Sutherland opened his own practice in Philadelphia while retaining a position at Children’s in the Obstetrics and Gynecology Department.
In 1928, Sutherland decided to set up practice in Brattleboro.